sci論文投稿cover letter 投稿信模版

sci論文投稿cover letter 投稿信模版

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時間:2017-10-22

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1、SCI論文投稿coverletter投稿信模版DeareditorialboardofEuropeanJournalofCardiology,Pleasefindenclosedthemanuscript:“Theangiotensin-convertingenzymeisnotariskfactorformyocardialinfarctioninFrenchindividuals”,bySarahH.,etal.,tobesubmittedasaShortCommunicationtotheEuropean

2、JournalofNeurologyforconsiderationofpublication.Allco-authorshaveseenandagreewiththecontentsofthemanuscriptandthereisnofinancialinteresttoreport.Wecertifythatthesubmissionisoriginalworkandisnotunderreviewatanyotherpublication.Inthismanuscript,wereporttheresu

3、ltsofthefirststudyonthegeneticandfunctionalrolesoftheACEontheriskofsufferingamyocardialinfarctionintheFrenchpopulation.Indeed,wegenotypedthers4341polymorphismin531IScasesand549healthycontrols,andthenperformedfunctionalstudiesbymeasuringserumACEproteinlevelan

4、dactivityinhealthycontrols,strokepatientsatbaselineandstrokepatients24hafterstrokesymptomsonset.TheresultsfromourstudydidnotrevealanyassociationoftheACEvariantwithmyocardialinfarction,althoughitaffectedACEproteinlevel,andischemicstrokepatientsshowedlowerACEl

5、evelthancontrolsintheacutephasebutnotinthechronicphase.WebelievethatourfindingscouldbeofinteresttothereadersofEuropeanJournalofCardiologybecausetheybringnewandstrongevidencethattheACEgeneandproteinarenotariskfactorformyocardialinfarction.Wehopethattheeditori

6、alboardwillagreeontheinterestofthisstudy.Sincerelyyours,SarahH.onbehalfoftheauthors.Correspondingauthor:SarahHamiltonatCardiovascularResearchLaboratory,MarieCurieResearchInstitute,75000,Paris,France,xxx@mariecurie.fr,phonenumber:+33582246xxx,faxnumber:+33582

7、246xxx.Case1?DearEditor,?Wewouldliketosubmittheenclosedmanuscriptentitled"GDNFAcutelyModulatesNeuronalExcitabilityandA-typePotassiumChannelsinMidbrainDopaminergicNeurons",whichwewishtobeconsideredforpublicationinNatureNeuroscience.?GDNFhaslongbeenthoughttobe

8、apotentneurotrophicfactorforthesurvivalofmidbraindopaminergicneurons,whicharedegeneratedinParkinson’sdisease.Inthispaper,wereportanunexpected,acuteeffectofGDNFonA-typepotassiumchannels,leadingto

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